By Vidya Bhushan Gupta
Impeccably researched and written through a nationally identified group of experts from neurodevelopmental pediatrics, psychology, epidemiology, schooling, occupational remedy, and speech remedy backgrounds, ''Autistic Spectrum problems in Children'' offers a multidisciplinary profile of autism and pervasive developmental problems in little ones. Tracing the heritage of autism and monitoring the newest study on its causation and neurological foundation, this quantity bargains either conventional clinical and complementary and substitute medication approaches-as good as trustworthy equipment for assessing therapy efficacy
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Additional resources for Autistim Spectrum Disorders in Children
4). The weighted and mean prevalence estimates among the studies that used population screening and direct Figure 4 Trend in prevalence rate for autism by number in population. Copyright © 2004 Marcel Dekker, Inc. 1/10,000, respectively. It is worth noting that studies that used the method of population screening and direct examination were also those that had smaller study populations. Therefore, this method may have been more plausible in such populations and, again, reinforces the notion that perhaps, it is possible to do a more intensive and comprehensive survey in these populations, leading to a greater number of individuals identiﬁed with autism or ASD.
The reasons for this are twofold. First, it is generally more difﬁcult to develop studies that measure the true incidence of autism because of inherent difﬁculties deﬁning newly occurring cases. The at-risk population consists of children typically diagnosed between 2 and 5 years of age although it is still not uncommon for older children and even adults to receive an initial ﬁrst ASD diagnosis (6). Further, the clinical diagnosis of ASD may have more to do with the ability and willingness to make an ASD diagnosis than with the true onset of pathology.
It is known from these studies, as well as others of chromosomal abnormalities and genetic conditions associated with autism, that there is undoubtedly a major genetic component to autism. One explanation consistent with this and the fact that no inheritance model appears to ﬁt the family data is that genetic and nongenetic factors may interact—with certain nongenetic factors increasing risk only when key predisposing genes are present. If this is the case, not only will future epidemiological studies investigating nonheritable autism risk factors need to be of larger size and stronger design, they will also need to be able to look for these factors within strata deﬁned by level of genetic susceptibility.